|
They also
declared categorically that they knew of no natural process by which one of
these two forms could have evolved into the other. According to the professional
science magazine, the fall 1984 annual meeting of the American Association for
the Advancement of Science (AAAS), was almost entirely devoted to the question
of: to what extent new pathogenic agents could be produced via human
manipulation of genes. According to the Segals, AIDS was practically the sole
topic of discussion. THE AIDS VIRUS The Segals discuss the findings of Gonda et
al, who compared the HIV, visna and other closely-related viruses and found that
the visna virus is the most similar to HIV. The two were, in fact, 60% identical
in 1986. According to findings of the Hahn group, the mutation rate of the HIV
virus was about a million times higher than that of similar viruses, and that on
the average a 10% alteration took place every two years.
That would mean that in
1984, the difference between HIV and visna would have been only 30%, in 1982-
20%, 10% in 1980 and zero in 1978. This means, say the Segals, that at this time
visna viruses changed into HIV, receiving at the same time the ability to become
parasites in human T4-cells and the high genetic instability that is not known
in other retroviruses. This is also consistent with the fact that the first
cases of AIDS appeared about one year later, in the spring of 1979. In his
comparison of the genomes of visna and HIV, add the Segals, Coffin hit upon a
remarkable feature. The env (envelope) area of the HIV genome, which encodes the
envelope proteins which help the virus to attach itself to the host cell, is
about 300 nucleotides longer than the same area in visna. This behaviour
suggests that an additional piece has been inserted into the genomes of the
visna virus, a piece that alters the envelope proteins and enables them to bind
themselves to the T4-receptors. BUT THIS SECTION BEHAVES LIKE A BIOLOGICALLY
ALIEN BODY, which does not match the rest of the system biochemically.
The above
mentioned work by Gonda et al shows that the HIV virus has a section of about
300 nucleotides, which does not exist in the visna virus. That length
corresponds with what Coffin described. That section is particularly unstable,
which indicates that it is an alien object. According to the Segals, it
originates in an HTLV-1 genome, (discovered by Gallo-ED) for the likelihood of
an accidental occurrence in HIV of a genome sequence 60% identical with a
section of the HTLV-1 that is 300 nucleotides in length is zero. Since the visna
virus is incapable of attaching itself to human T4 receptors, it must have been
the transfer of the HTLV-1 genome section which gave visna the capability to do
so. In other words, the addition of HTLV-1 to visna made the HIV virus.
In
addition, the high mutation rate of the HIV genome has been explained by another
scientific team, Chandra et al, by the fact that it is a combination of two
genome parts which are alien to each other BY ARTIFICIAL MEANS rather than by a
natural process of evolution, because this process would have immediately
eliminated, through natural selection, systems that are so replete with
disorders. These are the facts of the case, say the Segals. HIV is essentially a
visna virus which carries an additional protein monomer of HTLV-1 that has an
epitope capable of bonding with T4 receptors. Neither Alizon and Montagnier nor
any other biologist know of any natural mechanism that would make it possible
for the epitope to be transferred from HTLV-1 to the visna virus. For this
reason we can come to only one conclusion: that this gene combination arose by
artificial means, through gene manipulation.
THE CONSTRUCTION OF HIV The
construction of a recombinant virus by means of gene manipulation is
extraordinarily expensive, and it requires a large number of highly qualified
personnel, complicated equipment and expensive high security laboratories.
Moreover, the product would have no commercial value. Who, then, ask the Segals,
would have provided the resources for a type of research that was aimed solely
at the production of a new disease that would be deadly to human beings? The
English sociologist Allistair Hay (as well as Paxman et al in A Higher Form of
Killing-ED), published a document whose authenticity has been confirmed by the
US Congress, showing that a representative of the Pentagon requested in 1969
additional funding for biological warfare research.
The intention was to create,
within the next ten years, a new virus that would not be susceptible to the
immune system, so that the afflicted patient would not be able to develop any
defense against it. Ten years later, in the spring of 1979, the first cases of
AIDS appeared in New York. Thus began a phase of frantic experimentation, say
the Segals. One group was working on trying to cause animal pathogens to adapt
themselves to life in human beings. This was done under the cover of searching
for a cure for cancer. The race was won by Gallo, who described his findings in
1975. A year later, Gallo described gene manipulations he was conducting. In
1980 he published his discovery of HTLV. In the fall of 1977, a P4 (highest
security category of laboratory, in which human pathogens are subjected to
genetic manipulations) laboratory was officially opened in building 550 of Fort
Detrick, MD, the Pentagon's main biological warfare research center.
|
